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  #1  
Old 02-02-2012
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GIT Suspected H Pylori Gastritis!

A 42 year old woman complains of burning pain in the upper middle region of her abdomen. The pain usually occurs about 2 hours after a meal and frequently awakens her at night. Antacids can usually relieve the pain. An x ray film reveals a typical duodenal ulcer identified as a discrete crater in the proximal portion of the duodenal bulb. because the woman does not have a H/o Nsaids use, the H pylori is assumed to be the main factor in the etiology. Which of the following is likely to be normal in this woman?

A) Basal acid output
B) Fasting serum gastrin
C) Gastrin response to meal
D) Maximal acid output
E) Parietal cell mass
F) Pepsin secretion
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Old 02-03-2012
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fasting serum gastrin
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Old 02-03-2012
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fasting serum gastrin it is
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Old 02-03-2012
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Help this is from UW!! I read this..

Answer is fasting serum gastrin..
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Old 02-03-2012
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Default why not gastrin response to meals

I narrow it down to gastrin response to meals and fasting gastrin output, but can't tell which one is abnormal.
Can anyone explain?
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Old 02-03-2012
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Idea! hope this helps..

Quote:
Originally Posted by patho2012 View Post
I narrow it down to gastrin response to meals and fasting gastrin output, but can't tell which one is abnormal.
Can anyone explain?
Fasting gastrin levels are normal because in H.pylori infection the mucosal layer breakdown is what actually causes the ulcers, there is no cause for increased levels of gastrin; where as if it were to be something like Zollinger Ellison then there would be an increase in the gastrin levels by the gastrinoma that would cause more acid secretion hence leading to ulcers..
Helping?? Or still confused??
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Old 02-04-2012
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Gastrin response to meals should be normal as well? whether there is Helico bacter pylori there or not? it has nothing to do with the vagal stimulation?
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Old 02-04-2012
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how does H.pylori affect all these options?
i thought it just gets into the mucus layer so that its protected from the acid.
i didnt think it increased or decreased acid secretion at all.
and E doesn't make sense to me as its pepsinogen thats secreted, not pepsin.

help :/
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Old 02-05-2012
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The correct answer is B

DU patients have normal fasting Serum Gastrin, but their response to a meal is increased. Acid suppresses gastric release less effectively in DU.
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