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Old 12-10-2012
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ACEIs causes vasoconstriction of afferent arterioles ??,PGs also released to cause vasodilatation of afferent arterioles,this aims to improve the blood flow into the glomerulus and improve the renal perfusion. ACEIs causes vasoconstriction of afferent arterioles
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Old 12-10-2012
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Originally Posted by riya rai View Post
ACEIs causes vasoconstriction of afferent arterioles ??,PGs also released to cause vasodilatation of afferent arterioles,this aims to improve the blood flow into the glomerulus and improve the renal perfusion. ACEIs causes vasoconstriction of afferent arterioles
Angiotensin 2 causes vasoconstriction of efferent arteriole increasing GFR
ACEI cause low Angiotensin 2 and vasodilation of efferent arterioles that decreases GFR
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Old 12-13-2012
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ACEIs causes vasoconstriction of Aferent arterioles and PGs causes vasodilation in this way improve the flow in glomeruls and improve the renal perfusion,how i cant undrstand this concept,they wil oposite the each other action what wil b the net efect

Last edited by riya rai; 12-13-2012 at 09:59 AM.
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Old 12-13-2012
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Originally Posted by riya rai View Post
ACEIs causes vasoconstriction of Aferent arterioles and PGs causes vasodilation in this way improve the flow in glomeruls and improve the renal perfusion,how i cant undrstand this concept,they wil oposite the each other action what wil b the net efect
Sounds like you have your concepts mixed up a bit.
http://circ.ahajournals.org/content/...expansion.html

Angiotension II causes vasoconstriction on the efferent and afferent.
Ace Inhibitors prevents formation of AG-II and thus dilate the efferent arteriole.
And the PGs do vasodilate the afferent.

The net effect of using an ACEI would cause a decrease in GFR, and thus decrease Filtration Fraction.

Thus causing diuresis.
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