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  #101  
Old 01-10-2013
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Please guys, if anyone happen to have offline nbmes please do send them to my email hockey4230@yahoo.com
Thanks in advance
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  #102  
Old 01-11-2013
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guys can u plz discuss RING lesions/conditions..
i ll start wid
P.falciparum - ring form
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  #103  
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Default Ring lesions...

Quote:
Originally Posted by nandish_m View Post
guys can u plz discuss RING lesions/conditions..
i ll start wid
P.falciparum - ring form
Ring enhancing lesions in brain:
Toxoplasmosis
metastasis
abscesses
AIDS lymphoma.
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  #104  
Old 01-11-2013
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What are the drugs that act on microtubules?
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  #105  
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Quote:
Originally Posted by neha_subh View Post
Ring enhancing lesions in brain:
Toxoplasmosis
metastasis
abscesses
AIDS lymphoma.
one more
ringed sideroblast in sideroblastic anemia
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  #106  
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Quote:
Originally Posted by hockey View Post
What are the drugs that act on microtubules?
vincristine/vinblastine
colchicine
paclitaxel
griseofulvin
mebendazole/thiabendazole
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  #107  
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Please guys, if anyone happen to have offline nbmes please do send them to my email hockey4230@yahoo.com
Thanks in advance
please visit www.scribd.com
download from there
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  #108  
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peak flow in right coronary artery

Question on T3 and T4

whats your's views about these 2 questions
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  #109  
Old 01-13-2013
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Star pharma: Discoloration

Red man syndrome: vancomycin (Histamine release)
Gray(ashen) baby syndrome: chloramphenicol (dec.glucuronyl transferase)
Gray man syndrome(blue pigmentation of skin"smurf skin"): Amiodarone
Red green discrimination: Ethambutol
yellow discrimination: Digoxin
Red orange metabolite in secretions: Rifampin
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Last edited by neha_subh; 01-13-2013 at 08:12 AM.
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  #110  
Old 01-13-2013
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is there any mnemonics or easier way to remember lysosomal storage diseases enzyme deficiency n substrate accumulated??
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  #111  
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Quote:
Originally Posted by neha_subh View Post
Red man syndrome: vancomycin (Histamine release)
Gray(ashen) baby syndrome: chloramphenicol (dec.glucuronyl transferase)
Gray man syndrome(blue pigmentation of skin"smurf skin"): Amiodarone
Red green discrimination: Ethambutol
yellow discrimination: Digoxin
Red orange metabolite in secretions: Rifampin
thank ypu..
but wot is red green/yellow discrimination?
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  #112  
Old 01-13-2013
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Quote:
Originally Posted by neha_subh View Post
Hi,
i am planning to take step 1 in mid of feb2013,have finished 50% uworld (20 blocks), have planned to complete uworld in mid of jan,after that i will go thru first aid 2times,i will try to cover mine weak areas from kaplan notes,and will do NBME.

well this is mine study plan, lets motivate and encourage each thru this journey who are also taking exame on feb2013..



Get ready for feb 2013 and lets kick some doors open and help each other to reach our destinies....

Good luck to you and all the best on your STEP 1 Neha_Subh
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  #113  
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Quote:
Originally Posted by neha_subh View Post
peak flow in right coronary artery

Question on T3 and T4

whats your's views about these 2 questions


T3 down regulates TRH and therefore inhibits TSH. TSH secretes T4 and T3. T4 is highest in populaton.; whereas, T3 is little. However, T4 converts into T3. T3 itself is more potent; meaning active compare to T4. Therefore, TRH and TSH are regulated by T3.

When T3 is low, TSH will secrete T4 and T3. T4 will convert to T3. T3 will raise up to normal levels.

When T3 is high, it will inhibit TSH. TSH will not secrete T4 and T3. T4 will not convert to T3. T3 levels will drop down to normal levels.


So the answer is T3.
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  #114  
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Quote:
Originally Posted by nandish_m View Post
guys i finished fa once, finished uw score- 50%
yesterday gave nbme n scored 165
my target is >230
need some motivation and advise
i can postpone till feb end..
thanx

Motivation: Don't give up easily. You will make it through. Good luck and all the best to you on your STEP 1.
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  #115  
Old 01-13-2013
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Default step 1 kaplan

hey does anybody here hav step 1 kaplan q bank offline?any body can provide it?
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  #116  
Old 01-14-2013
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Quote:
Originally Posted by Doctor Ali View Post
T3 down regulates TRH and therefore inhibits TSH. TSH secretes T4 and T3. T4 is highest in populaton.; whereas, T3 is little. However, T4 converts into T3. T3 itself is more potent; meaning active compare to T4. Therefore, TRH and TSH are regulated by T3.

When T3 is low, TSH will secrete T4 and T3. T4 will convert to T3. T3 will raise up to normal levels.

When T3 is high, it will inhibit TSH. TSH will not secrete T4 and T3. T4 will not convert to T3. T3 levels will drop down to normal levels.


So the answer is T3.
but what physio kaplan lectures notes says: T3 levels doesnt efects the TSH levels no mater whats its concntration in blood increase or decrease but when significant changes to T4 levels wil efects the TSH levels,and even when i did online kaptest (qbank) that option was correct T4 not T3
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  #117  
Old 01-14-2013
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Quote:
Originally Posted by nandish_m View Post
thank ypu..
but wot is red green/yellow discrimination?

Deuteranopia is a color vision deficiency in which the green retinal photoreceptors are absent, moderately affecting red–green hue discrimination.(color blindness)

http://en.wikipedia.org/wiki/Color_blindness

http://www.ncbi.nlm.nih.gov/pubmed/8447096
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  #118  
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Quote:
Originally Posted by neha_subh View Post
peak flow in right coronary artery

Question on T3 and T4

whats your's views about these 2 questions
der is a line 4m kaplan book..
As long as ciculating free T4 remains normal, changes in circulating T3 have minimal effect on TSH secretion.
In d question,we r talking abt healthy person(normal T4).. so we rule out exogenous T3.

now because the main circulationg form is T4, it is T4 that is responsible for most of negative feedback at the level of antr pitutary.(KAPLAN PHYSIO)

so i.v T4 --> DECRESE TSH ---> DECRESE T4

ans - T4
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  #119  
Old 01-14-2013
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Can someone explain Confounding bias and Effect modification..? Too confusing!
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  #120  
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Originally Posted by usmleprep_sr View Post
Can someone explain Confounding bias and Effect modification..? Too confusing!
Confounding- exposure-disease is mixed. Lung cancer and alcohol concumption and than compare smoking and non-smoking.

effect of modification- exposure of interest an outcome is modified by another factor. ex-oral contraception use in breast cancer modify by family history.

I'm using UW biostatistics is really good!
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  #121  
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Quote:
Originally Posted by neha_subh View Post
but what physio kaplan lectures notes says: T3 levels doesnt efects the TSH levels no mater whats its concntration in blood increase or decrease but when significant changes to T4 levels wil efects the TSH levels,and even when i did online kaptest (qbank) that option was correct T4 not T3


This is very interesting. Some say T3 whereas others will say T4. So where is this problem? Where is this confusion? I will do my best to explain the Thyroid hormone topic. At the end and hopefully the confusion will be over.


Here’s what happens:

1. TRH is secreted by the hypothalamus and stimulates the secretion of TSH by the anterior pituitary.

2. TSH increases both synthesis and secretion of Thyroid hormones, T3 and T4.

3. T4 converts into T3 and rT3.

4. rT3 is inactive form.

5. More T4 is synthesized than T3. In other words, T4 >>>> T3.

6. However, T3 is more biologic active than T4. In other words, T3 is more potent than T4. In other words, T3 >>>> T4.



With this in mind, we all know the functions/ actions of Thyroid hormone right? The 4 B’s – Brain maturation, bone growth, BMR, and Beta adrenergic effects. All of these actions are done by T3.


Therefore, down – regulation, meaning, negative feedback, is done by T3. T3 down – regulates TRH receptors in the anterior pituitary and thereby inhibits TSH secretion. If you give exogenesis T3 to a patient for example, what will be the result? Answer: Decrease TSH, decrease T4, decrease rT3, and Increase T3.

In this question, it’s asking similar way:
In a healthy patient, an injection of which substance would DECREASE the patient's T4 levels?

A. Thyroglobulin
B. T3
C. T4
D. Reverse T4


If we look at the question very carefully, the question is really asking which substance would decrease the T4 levels. And that substance is injected, meaning, exogenesis; not endogenesis.

T4 is another name for Levothyroxine. Levothyroxine is use for increasing thyroid hormones, T3 and T4.


By looking this way, I hope that the troubling and confusion will be cleared out.


My sources are:
1. FA 2012 pg 322
2. BRS Physiology 4th ed. Pg 247
3. BRS Physiology 5th ed. Pg 243
4. UW
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  #122  
Old 01-15-2013
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Quote:
Originally Posted by rahul usmle View Post
hey does anybody here hav step 1 kaplan q bank offline?any body can provide it?
yes any one plzzzz......
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  #123  
Old 01-15-2013
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Default mouth/hand/feet diseases:

Quote:
Originally Posted by vnagubandi View Post
Diseases/Lesions with palms and soles involvement

Secondary Syphilis = maculopapular rash on palms and soles
Coxsackie Virus A (hand,foot and mouth disease)
Rocky mountain Spotted Fever (Rickettsial rash) (Imp.points typhus rash starts centrally and spreads outwards without involving palms and soles)
Painful red lesions on palms and soles = Osler's node (Infective endocarditis)
Painless erythematous lesions on palms and soles+splinter hemorhages in finger nails = Janeway lesions (infective endocarditis)

Mnemonic We drive CARS with Palms and Soles (Cox A,Rocky and Syphilis) rest we ca remember painful osler's painless janeway
add more:
painful blue fingers/toes,hemolytic anemia: cold aglutination disease (autoimune hemolytic anemia due to mycoplasma pneumoniae ,infectious mononucleosis)
painful,pale,cold fingers/toes: raynaud's syndrome (vasospasm in extremities)
small irregular red spots on buccal/lingual mucosa with blue white centers: koplick spots(measles).
hyperpigmentation of mouth/feets/hands: peutz jehgers syndrome
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  #124  
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Default First aid Discoloration:

Quote:
Originally Posted by neha_subh View Post
Red man syndrome: vancomycin (Histamine release)
Gray(ashen) baby syndrome: chloramphenicol (dec.glucuronyl transferase)
Gray man syndrome(blue pigmentation of skin"smurf skin"): Amiodarone
Red green discrimination: Ethambutol
yellow discrimination: Digoxin
Red orange metabolite in secretions: Rifampin
Blue sclera: osteogenesis imperfecta
Bluish line on gingiva(burton's line): lead poisoning
Blue painful fingers: cold aglutination disease
pale painful cold fingers toes: raynauds syndrome
Red painful raised leisons on palms/soles: oslers nodes(rheumatoid arthritis)
Red painles on palms/soles: janeway lesions(inf.endocarditis)
Dark Purple skin/mouth nodules: kaposi sarcoma
Green yellow rings around peripheral cornea: kayser-fleisher rings
Red "curant jelly sputum" in alcoholics or diabetic patients: Klebsiela pneumoniae
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  #125  
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Default pulmonary emboli:

Q: pulmonary emboli:
what changes would you expect?

PAO2: inc/dec?
pACO2: inc/dec?
PaO2: inc/dec?
PaCO2: inc/dec?
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  #126  
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Quote:
Originally Posted by eveli55 View Post
Confounding- exposure-disease is mixed. Lung cancer and alcohol concumption and than compare smoking and non-smoking.

effect of modification- exposure of interest an outcome is modified by another factor. ex-oral contraception use in breast cancer modify by family history.

I'm using UW biostatistics is really good!
i have BBB:Biostatics (Behavioral science) Brain Barrier
thats why am worried about these bariers i tried alot to improve biostatic but uw score is not improving
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  #127  
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Quote:
Originally Posted by neha_subh View Post
Blue sclera: osteogenesis imperfecta
Bluish line on gingiva(burton's line): lead poisoning
Blue painful fingers: cold aglutination disease
pale painful cold fingers toes: raynauds syndrome
Red painful raised leisons on palms/soles: oslers nodes(rheumatoid arthritis)
Red painles on palms/soles: janeway lesions(inf.endocarditis)
Dark Purple skin/mouth nodules: kaposi sarcoma
Green yellow rings around peripheral cornea: kayser-fleisher rings
Red "curant jelly sputum" in alcoholics or diabetic patients: Klebsiela pneumoniae
Both osler and janeway lesions are seen in IE..Osler nodes are painfull and on dorsaL side of fingers,toes ..Janeway are painless and on palms and sole..
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  #128  
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Quote:
Originally Posted by usmleprep_sr View Post
Both osler and janeway lesions are seen in IE..Osler nodes are painfull and on dorsaL side of fingers,toes ..Janeway are painless and on palms and sole..
oh ya sorry,you are right thanks for correction
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Quote:
Originally Posted by neha_subh View Post
Q: pulmonary emboli:
what changes would you expect?

PAO2: inc/dec?
pACO2: inc/dec?
PaO2: inc/dec?
PaCO2: inc/dec?
Pulmonary emboli: Inc V/Q mismatch due to dec perfusion or Q. Hypoxemia->hypervent->resp alkalosis..
Hence PH inc,Paco2 dec,pao2 dec..I guess PA o2 and PA co2 shud be normal..
Correct me if im wrong..
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  #130  
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Default saddle nose...

saddle nose is seen in folowing conditions: CLAW


congenital syphilis
cocaine abuse
leprosy
Achondroplasia (relapsing polychondritis)
wegeners granulomatosis

www.en.wikipedia.org/wiki/saddle_nose
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  #131  
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Question biochem: questions

give liltle clues regarding:

Refsum disease cause?
zelweger syndrome cause?
polysomes what it is?
proteosomes function?
peroxisomes function?
lysosomes function?
lysozymes function?
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  #132  
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Default pulmonary emboli...

Quote:
Originally Posted by usmleprep_sr View Post
Pulmonary emboli: Inc V/Q mismatch due to dec perfusion or Q. Hypoxemia->hypervent->resp alkalosis..
Hence PH inc,Paco2 dec,pao2 dec..I guess PA o2 and PA co2 shud be normal..
Correct me if im wrong..

pulmonary emboli:

PAO2: increased
PACO2: decreased
PaO2: decreased
PaCO2: decreased
PH: Increased
Increase VA/Q ratio

acording to kaplan physio notes: pulm.emboli stucked in pulmonary artery and ultimately dec left sided heart flow--decrease cardiac output--increased Heart rate---- due to hypoxemia(dec.PaO2)--vagal stimulation in lungs-- increased respiratory rate--respiratory alkalosis--dec.PaCO2
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  #133  
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Quote:
Originally Posted by neha_subh View Post
pulmonary emboli:

PAO2: increased
PACO2: decreased
PaO2: decreased
PaCO2: decreased
PH: Increased
Increase VA/Q ratio

acording to kaplan physio notes: pulm.emboli stucked in pulmonary artery and ultimately dec left sided heart flow--decrease cardiac output--increased Heart rate---- due to hypoxemia(dec.PaO2)--vagal stimulation in lungs-- increased respiratory rate--respiratory alkalosis--dec.PaCO2
but here am confused about PaO2 and PaCO2, if pulmonary arterial blood not going to lungs for oxygenation then PaCO2 concentration should be raised why its going to be decreased
ok lets supose if PaCO2 concentration is going to be decreased inspite of blood is not going into lungs for oxygenation then PaO2 concentration should also be increased not decreased
i forgot its concept pls explain it :sorry:
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  #134  
Old 01-15-2013
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Default Functional organization of the gene

Can some tel me where are the Promoter Enhancer located on the gene ,,,, its on 3 prime end or 5 prime end ??????
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  #135  
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Drug induced Pulmonary Fibrosis:

Bleomycin
Busulfan
Amiodarone
Tocainide
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Quote:
Originally Posted by mymleprep View Post
Can some tel me where are the Promoter Enhancer located on the gene ,,,, its on 3 prime end or 5 prime end ??????
Promoter is on 3'
Enhancer can be at 3' many base pairs ahead, in gene intron, or at 5' many base pairs downstream
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Drug induced lupus:

Hydralazine
Isoniazid
Procainamide
Phenytoin
Penicillamine
Ethosuximide
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Thank you... was confused reading 2010 and 2012 FA

Sent from my GT-I9100 using Tapatalk 2
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Quote:
Originally Posted by mymleprep View Post
Can some tel me where are the Promoter Enhancer located on the gene ,,,, its on 3 prime end or 5 prime end ??????
Promoter is located on 5 prime end of DNA..There is FA 2012 error which says 3 prime..
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Quote:
Originally Posted by neha_subh View Post
pulmonary emboli:

PAO2: increased
PACO2: decreased
PaO2: decreased
PaCO2: decreased
PH: Increased
Increase VA/Q ratio

acording to kaplan physio notes: pulm.emboli stucked in pulmonary artery and ultimately dec left sided heart flow--decrease cardiac output--increased Heart rate---- due to hypoxemia(dec.PaO2)--vagal stimulation in lungs-- increased respiratory rate--respiratory alkalosis--dec.PaCO2
How are Alveolar conc changed? i.e PA o2 and PA co2...??? There is no ventilation defect and shunt.. Can someone explain..
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Quote:
Originally Posted by usmleprep_sr View Post
How are Alveolar conc changed? i.e PA o2 and PA co2...??? There is no ventilation defect and shunt.. Can someone explain..
ya its kaplan physio concept thats why she has asked about this ,am also confused bw uw and kaplan notes concepts
as you said dec PaO2 and dec PaCO2 same uw concept but kaplan physio concept: dec PACO2 & inc.PAO2 and inc.PH. (cofirm it from physio kaplan lectures notes resp.system chapter 4)
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Quote:
Originally Posted by usmleprep_sr View Post
Promoter is located on 5 prime end of DNA..There is FA 2012 error which says 3 prime..
Sorry..Its 3 prime..Though there is FA 12 error in marking gene 5 prime to 3 prime..TATTT shud be at 3 prime(Referred FA errata)
If you look at pic in kaplan,promoter is seen in any direction on ds dna.But it precedes transcription at 3 prime of template strand..The other strand is in 5 prime to 3 prime direction called coding/anti template strand..
plz correct me if im still wrong.
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Default infections which involves the cranial or spinal nerves:

Q: Name the infections which involves the cranial/spinal nerves??
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Default conditions which afects the teeth....

Q:Name the conditions which afects the teeth???
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Quote:
Originally Posted by neha_subh View Post
give liltle clues regarding:

Refsum disease cause?
zelweger syndrome cause?
polysomes what it is?
proteosomes function?
peroxisomes function?
lysosomes function?
lysozymes function?
Refsum disease and zelweger syndrome are the examples of peroxisomes diseases..
Peroxisomes: where long chain and branched chain(odd no.carbons) faty acids oxidized...
proteosomes: degrades the improper or un needed intracelular proteins
lysosomes: degrades the faty acids,CHO,protein and nucleic acids
lysozymes: antimicrobial enzyme present in body secretions (tears,saliva,human milk and mucus) and in cytoplasm of neutrophils.
polysomes (polyribosomes or ergosomes): are the clusters of ribosomes bound to mRNA and read one mRNA simultaneously.
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Q:Name the conditions which afects the teeth???
syphilis: hutchinsons incisors,mulbery molars
lead poisoning: blue line along the gum,bluish blue edging to the teeth known as Burton line seen with chronic lead poisoning,



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Save It! Here r some

Quote:
Originally Posted by neha_subh View Post
syphilis: hutchinsons incisors,mulbery molars
lead poisoning: blue line along the gum,bluish blue edging to the teeth known as Burton line seen with chronic lead poisoning,



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OSteogenesis imperfecta - lack dentin
Tetracycline - blue discoloration
Job's syndrome - primary tooth retention
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Quote:
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Q: Name the infections which involves the cranial/spinal nerves??
syphilis: involves the 8TH cranial nerve.teritiary syphilis: tabes dorsalis(dorsal roots and dorsal column degenration)
HSV 1: 5TH cranial ganglia
HSV 2: sacral ganglia
VZV: (5TH)trigeminal and dorsal root ganglia
polio: lower motor neuron lesion
Borellia bugdorferi (stage 2): 7th cranial nerve (bell's palsy)
Campylobacter: GBS (Demyelination of peripheral nerves and motor fibers of ventral roots)

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syphilis: involves the 8TH cranial nerve.teritiary syphilis: tabes dorsalis(dorsal roots and dorsal column degenration)
HSV 1: 5TH cranial ganglia
HSV 2: sacral ganglia
VZV: (5TH)trigeminal and dorsal root ganglia
polio: lower motor neuron lesion
Borellia bugdorferi (stage 2): 7th cranial nerve (bell's palsy)
Campylobacter: GBS (Demyelination of peripheral nerves and motor fibers of ventral roots)
Rubella virus (togavirus): 8th cranial nerve (sensory neural deafness in fetus)
vitamin B6: Peripheral neuropathy (vit B6 inducible by INH and oral contraceptives)
vitamin B12: Subacute combined degenration,paresthesias
vitamin E: Posterior column and Spinocerebelar tract demyelination,muscle weaknes.

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  #150  
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Quote:
Originally Posted by neha_subh View Post
syphilis: involves the 8TH cranial nerve.teritiary syphilis: tabes dorsalis(dorsal roots and dorsal column degenration)
HSV 1: 5TH cranial ganglia
HSV 2: sacral ganglia
VZV: (5TH)trigeminal and dorsal root ganglia
polio: lower motor neuron lesion
Borellia bugdorferi (stage 2): 7th cranial nerve (bell's palsy)
Campylobacter: GBS (Demyelination of peripheral nerves and motor fibers of ventral roots)

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Rabies via ACh receptor- travel retrograde
GBS also caused by HSV (FA2012)
HSV causes Ramsay hunt- facial paralysis
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Quote:
Originally Posted by riya rai View Post
ya its kaplan physio concept thats why she has asked about this ,am also confused bw uw and kaplan notes concepts
as you said dec PaO2 and dec PaCO2 same uw concept but kaplan physio concept: dec PACO2 & inc.PAO2 and inc.PH. (cofirm it from physio kaplan lectures notes resp.system chapter 4)

in PE there is dyspnea resulting in hyperventilation ;

hyperventilation co2 washed out causing inc in PAo2 and pH
so paco2 dec and since some respiratory units r not perfused no gas exchange takes place in them dec pao2 ; hyperventilation doesnt compensate fully resulting in pao2 dec
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Can sum1 explain:

What will be the compliance of lungs if they are inflated with saline fluid?
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Quote:
Originally Posted by neha_subh View Post
i have BBB:Biostatics (Behavioral science) Brain Barrier
thats why am worried about these bariers i tried alot to improve biostatic but uw score is not improving
this is complicated subject. Just need time to understand . Also need review it everytime. But biostatistics is not a main subject on USMLE.
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Quote:
Originally Posted by billy View Post
Can sum1 explain:

What will be the compliance of lungs if they are inflated with saline fluid?
compliance will increases.
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Angry UW

Anybody could tell me what should i score in UW to be in safe side please??????
I can only do FA for another time and go for exam.
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Default Pls Guide me

hello everyone, my exam is on feb 21. My 15-20% of u world is left.. As one month is left what should be good for me.

1) Complete rest of u world asap simultaneously keep on revising my review book and after that when once u world is over in rest of the time say 15 to 20 days do u world 2 time (2 blocks a day).In this situation i would be able to go through only 40-50% of u world

2) From tomorrow onward do u wrld(2 blocks/day) complete in 24 days and simultaneously keep reading my review book.In this situation left over question would also be covered.
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Quote:
Originally Posted by billy View Post
Can sum1 explain:

What will be the compliance of lungs if they are inflated with saline fluid?
i think compliace is lost,i cant explain it but somewhere on internet i have read this article
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Anybody could tell me what should i score in UW to be in safe side please??????
I can only do FA for another time and go for exam.
i cant tel u about safe side uw score but do FA multiple times + at least one online NBME for assesing prep.+ asesment tests
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Quote:
Originally Posted by DrNitinArya View Post
hello everyone, my exam is on feb 21. My 15-20% of u world is left.. As one month is left what should be good for me.

1) Complete rest of u world asap simultaneously keep on revising my review book and after that when once u world is over in rest of the time say 15 to 20 days do u world 2 time (2 blocks a day).In this situation i would be able to go through only 40-50% of u world

2) From tomorrow onward do u wrld(2 blocks/day) complete in 24 days and simultaneously keep reading my review book.In this situation left over question would also be covered.
it seems to be good schedule,do FA multiple times+do Nbme + asesment tests
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Quote:
Originally Posted by vnagubandi View Post
This is a big one

African-American it is kind of like a buzzword. I mean what are all the things associated with African americans? (Diagnosis,Disorders,traits,associations,incidence ....anything). If we discuss it here i think we can group them all in one place and can get an idea of what to think about during the exam when we see "African American".

I mean NO OFFENSE to anyone. It is just to group under one heading the various things we see on the exam and for easier recall. I am just asking this because i've seen it on a lot of questions repeatedly.

Sickle cel disease
G6PD-deficiency
a-thalasemia
sarcoidosis
shistosomiasis hematobium infection

while b-thalasemia seen in mediteranean population
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Default Fresh water....

infections/conditions asociated with fresh water:

Diphylobothrium latum: larvae from raw fresh water fish--leads vitamin B12 deficiency

schistosoma: larvae in freshwater habitat of snails: leads intestinal,urinary,hepatic schistosomiasis


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Quote:
Originally Posted by neha_subh View Post
infections/conditions asociated with fresh water:

Diphylobothrium latum: larvae from raw fresh water fish--leads vitamin B12 deficiency

schistosoma: larvae in freshwater habitat of snails: leads intestinal,urinary,hepatic schistosomiasis


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drinking water- drcunculus medinensis
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  #163  
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Default Fresh water

Quote:
Originally Posted by neha_subh View Post
infections/conditions asociated with fresh water:

Diphylobothrium latum: larvae from raw fresh water fish--leads vitamin B12 deficiency

schistosoma: larvae in freshwater habitat of snails: leads intestinal,urinary,hepatic schistosomiasis


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Mountain climbing and water drinking: Giardia
HAV also by driking contaminated water
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  #164  
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Quote:
Originally Posted by neha_subh View Post
infections/conditions asociated with fresh water:

Diphylobothrium latum: larvae from raw fresh water fish--leads vitamin B12 deficiency

schistosoma: larvae in freshwater habitat of snails: leads intestinal,urinary,hepatic schistosomiasis


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PARASITES
T.solium - cysticercosis (swiss cheese mass lesion in brain)
E.granulosus - hydatid cyst(if ruptured ---> anaphylaxis)
Ascariasis
E.vermicularis - is pinworm, anal pruritis


BACTERIA
Botulism
Cholera
Dysentry
Ligeonella
M.marinum in immunocompromised
Salmonellosis/ typhoid

Plz correct me if i am wrong..
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Default Pls Guide me

hello everyone, my exam is on feb 21. My 15-20% of u world is left.. As one month is left what should be good for me.

1) Complete rest of u world asap simultaneously keep on revising my review book and after that when once u world is over in rest of the time say 15 to 20 days do u world 2 time (2 blocks a day). In this situation i would be able to go through only 40-50% of u world

2) From tomorrow onward do u wrld (2 blocks/day) complete in 24 days and simultaneously keep reading my review book. In this situation left over question would also be covered.
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Old 01-21-2013
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Default Brazilians!

Hi there, I would like to get in touch with any brazilians that are taking usmle.
I have already done step 2 , and will do step 1 in a couple of months...
My goal is to find some more brazilians out there, so we could work and succeed together!
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Default MI treatment:

MI treatment: SANBHA

Suplemental O2:for ischemia
ACEIs: dec.the after load + left vent.dilatation
Nitrates: dec.preload
Beta blockers: dec. the risk for arythmia
Heparin: limits the thrombosis formation
Aspirin: limits the thrombosis formation
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  #168  
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hey guys
taking exam on feb 6...anyone else taking around that time?
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Can some explain Mosaicism,Linkage disequilibrium with examples ....Not getting the concept of Mosaicism at all...
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Quote:
Originally Posted by dr.hobbes View Post
hey guys
taking exam on feb 6...anyone else taking around that time?
hey hobby me too on 6th feb... yesterday gave online nbme form 7 and scored 235 .... can u plz share ur nbme scores ... thanks
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aiming for 250+ .... yesterday gace nbme form 7 online and scored 235(560) ... high performance with star in all fields except behavioaral sciences and general prnciples of health and diseases .... plz advice how to improve it... aiming to give test on feb 6 ...
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Old 01-23-2013
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Nbme 11 231
Nbme 12 233....1 wk back
I have borderline performance in genetics and bs
Doing fa and uworld, Kaplan for genetics
I don't know Wht to do with bs
I hope I can improve
Suggestions ?
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Quote:
Originally Posted by Wajahat View Post
aiming for 250+ .... yesterday gace nbme form 7 online and scored 235(560) ... high performance with star in all fields except behavioaral sciences and general prnciples of health and diseases .... plz advice how to improve it... aiming to give test on feb 6 ...
I guess you can give Conrad ethics a read... I Haven't done tht yet... Practice biostat frm qbank... Did u revise uworld?
I am doing uworld 2nd time now ... I plan on finishing tht in a few days and give another nbme
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Quote:
Originally Posted by dr.hobbes View Post
I guess you can give Conrad ethics a read... I Haven't done tht yet... Practice biostat frm qbank... Did u revise uworld?
I am doing uworld 2nd time now ... I plan on finishing tht in a few days and give another nbme
yes i have done UW two times.... random timed mode and finished on 82 percent.... tryig to find 100 cases by conrad ... if u have them in pdf form kindly send it to me at wajahat_humayun@yahoo.com


thanks
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  #175  
Old 01-23-2013
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Quote:
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yes i have done UW two times.... random timed mode and finished on 82 percent.... tryig to find 100 cases by conrad ... if u have them in pdf form kindly send it to me at wajahat_humayun@yahoo.com


thanks
check ur inbox.. i have mailed u 100 cases..
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  #176  
Old 01-24-2013
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Quote:
Originally Posted by nandish_m View Post
check ur inbox.. i have mailed u 100 cases..
bundle of thanks ....
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  #177  
Old 01-24-2013
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Default NBME

I need the NBME's????????
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  #178  
Old 01-24-2013
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Hey there
I have exam in feb too.
Just wana know if u guys are revising kaplan at this moment?
I mean last look?
Not sure if its worth
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  #179  
Old 01-24-2013
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I am planning on doing kaplan q bank instead of books along with FA.
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  #180  
Old 01-26-2013
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Default Breast tumors:

BENIGN:
Fibroadenoma: MC tumor in less than 25yrs age,
small,mobile,firm with sharp edges mas
not inc.risk of CA
Intraductal papiloma:serous/bloody niple discharge
smal tumor beneath the areola
inc.risk of CA
Phylodes tumor: MC in 6th decade
Leaf like projection of tumor
large mass of conective tisue+cysts
MALIGNANT:
Ductal CA in situ:No BM involvement
arises from ductal hyperplasia+fils the ductal lumen
Invasive lobular: Bilateral.orderly row of cels
Inavasice ductal:Rock hard,firm,fibrous
MeduLLary: Lymphocytic infiltrate,ceLLular,fLeshy
Comedocarcinoma:ductal, Caseous necrosis,
Pagets desease: Eczema on niples
(Pagets cels: large cels with clear halo)
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  #181  
Old 01-26-2013
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Help

Quote:
Originally Posted by nandish_m View Post
check ur inbox.. i have mailed u 100 cases..
hi, can u plz send me to plz....
All the best for u
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  #182  
Old 01-26-2013
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Quote:
Originally Posted by Medman11 View Post
I need the NBME's????????
hey, do u hav a drop box account , i can share u on dat
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  #183  
Old 01-26-2013
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hi, me to planing in Feb. kaplan read 1 time , thn only u wrld 2 times. starting FA today! confused to do kaplan Q bank beside reading FA? any suugestions plz:sorry:

also any one knws minimum hw early we have to schedule our test? eg if i want to give on 28 feb hw early shld i shedule?

Last edited by gen88; 01-26-2013 at 03:11 AM.
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  #184  
Old 01-26-2013
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Quote:
Originally Posted by gen88 View Post
hey, do u hav a drop box account , i can share u on dat
ya i do have.. what do i have to provide u to share it?
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  #185  
Old 01-26-2013
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Quote:
Originally Posted by nandish_m View Post
ya i do have.. what do i have to provide u to share it?
can u plz email me saeedafatima88@yahoo.com or if u share on drop box. thanks
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  #186  
Old 01-28-2013
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Default variable expression:

variable expresion: ocurs in which modes of inheritance and its cause
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  #187  
Old 01-28-2013
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Quote:
Originally Posted by mymleprep View Post
Can some tel me where are the Promoter Enhancer located on the gene ,,,, its on 3 prime end or 5 prime end ??????
promoter located upstream and enhancer can be anywhere: downstream or upstream.
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  #188  
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Quote:
Originally Posted by neha_subh View Post
variable expresion: ocurs in which modes of inheritance and its cause
autosomal dominant inheritance. Examples are Neurofibromatosis 1 , MArfan syndrome, Hereditary hemochromatosis.
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  #189  
Old 01-28-2013
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Quote:
Originally Posted by eveli55 View Post
autosomal dominant inheritance. Examples are Neurofibromatosis 1 , MArfan syndrome, Hereditary hemochromatosis.
also ocurs with mitochondrial inheritance cause of that is heteroplasmy (presence of normal+mutated mitochondria)
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  #190  
Old 01-28-2013
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Default african-american conditions:

Quote:
Originally Posted by neha_subh View Post
Africa-american conditions:
Sickle cel disease
G6PD-deficiency
a-thalasemia
sarcoidosis
shistosomiasis hematobium infection

while b-thalasemia seen in mediteranean population
salmonela typhi and paratyphi seen in asia,africa,south america
chlamydia trachomatis: seen in africa-america
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  #191  
Old 01-28-2013
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Default amiodarone:inc.prolactin levels

amiodarone indirectly increases the prolactin levels by causing the hypothyroidism--there wil be no -ve feedbcak--inc:TRH---Inc: prolactin
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  #192  
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mechanism of tetany in Alkalosis?
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  #193  
Old 01-28-2013
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Default Alkalosis and tetany

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mechanism of tetany in Alkalosis?

In alkalosis there is decrease in protons (H) and increase in negativity of albumin. The more negative the albumin more it binds to Ca. Total Ca is normal but ionised or free will be reduced leading to tetany.
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  #194  
Old 01-28-2013
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Default African american

Quote:
Originally Posted by neha_subh View Post
salmonela typhi and paratyphi seen in asia,africa,south america
chlamydia trachomatis: seen in africa-america
Schstosoma is is common in egypt and not in african americans. Also alpha thalassemia is more common on southeast of asia that also increases risk of choriocarcinoma due to high pregnancy loss. chlamydia trachomatous Type A,B and C causes african blindness not african american.
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  #195  
Old 01-29-2013
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Default Am i ready ????

nbme 7 online a week before= 235
uwsa-1 today= 245

target is anything above 240
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  #196  
Old 01-29-2013
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Quote:
Originally Posted by Wajahat View Post
nbme 7 online a week before= 235
uwsa-1 today= 245

target is anything above 240
have heard uwsa overpredicts.. wen u planning to give ur exams?
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  #197  
Old 01-29-2013
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Don't go with uworl prediction ... Give nbme 11 or 12... And then decide
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  #198  
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Quote:
Originally Posted by dr.hobbes View Post
Don't go with uworl prediction ... Give nbme 11 or 12... And then decide
ok thanks ... i will inform u after giving my nbme
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  #199  
Old 01-29-2013
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Quote:
Originally Posted by Medman11 View Post
Schstosoma is is common in egypt and not in african americans. Also alpha thalassemia is more common on southeast of asia that also increases risk of choriocarcinoma due to high pregnancy loss. chlamydia trachomatous Type A,B and C causes african blindness not african american.
alpha thalesemia:asian and african population (first aid)
chlamydia trachomatis:serotypes L1,2,3 prevalent in africa,south america,asia (kaplan lecture notes)
schistosoma: acording to kaplan its comon in egypt but what uw says its comon in african-american population
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  #200  
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Mst common lesion a/w asbestos - FIBROUS PLAQUE OF PLEURA

Mst common cancer a/w asbestos- Primary lung cancer
2nd mst common - mesothelioma


occupations a/w asbestos exposure- roofers, persons workin in ship yard, brake lining workers
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