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Old 06-19-2013
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Heart LV afterload increased in AR

In a question i came across this point in which it says.. LV After load is already increased in Aortic Regurgitation caused by Infectious endocarditis.
Can some one show some light how did after load is already increased in AR and how does INFECTIVE ENDOCARDITIS CAUSE AR?
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Old 06-20-2013
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Infective endocarditis ----> vegetations on the valve leaflets --> degeneration of the Valve leaflet --> insufficiency of the valve --> regurgitation of the blood --> blood pools in the left ventricle --> heart works more to overcome the increased preload and ultimately this results in increased syestemic BP --> Increased afterload
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Old 06-21-2013
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but isnt afterload the resistance that the systemic vessels exert on the heart? How can the resistance increase due to AR?
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Quote:
Originally Posted by water View Post
but isnt afterload the resistance that the systemic vessels exert on the heart? How can the resistance increase due to AR?
Afterload is the tension or stress developed in the wall of the left ventricle during ejection.... in simple words the force against which the heart contracts

laplace law states Tension = PR/T

3 main factors affecting it are diastolic BP of aorta , diameter of the ventricle when contracting and the thickness of the muscle wall

as blood regurgitates in AR we have more blood pooling in the left ventricle and its diameter increase .... dilated heart must create a greater tension to overcome the same aortic pressure to eject blood hence increasedafterload.
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Old 06-21-2013
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Nice explanation!!
Thank you!!
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