I have studied immunology from Levinson and I found it really easy.
But now when i read it from Kaplan its too lengthy. And I personally think no ones gonna ask us such small details. is that so? Please help me out.
The questions also I cant solve them too deep. Should I be using some other book?
Just want to confirm something..
is the mutation of CRCX4 will give you a FULMINANT HIV Course? i suppose cause directly will get to Th4 and no macrophage, CTL or NK help from them...? please enlighten me! a friend of mine got this question about mutation on CRCX4 :confused:
What's the mechanism behind the development of sacroilitis, ankylosing spondylitis etc in Crohn's disease? Is it a hypersensitivity reaction? due to antibody/immune complex mediated damage of the joint? Is p-ANCA or any other antibody responsible for all these extraintestinal manifestations...
The advantages of breastfeeding:
• Passive transfer of T-cell immunity: Decreased risk of allergies and gastro*
intestinal and respiratory infections (MTB3-p.339/KLN Pediatrics-p36)
is it the IgA or is it the Tcell immunity playing a role ?
can someone please advice me , how to improve the subject above , i did a stimulated test , and i scored badly for the above two subject , i have used kaplan , dit , but still ..... please advice , thanks .
Ok, this one is probably a very overlooked topic but a very High Yield one, so lets start a discussion and postings of useful links and Vids explaining USMLE Lab Techniques found in Genetics/Immuno/Biochem etc. ( PCR, Blots, Assays, Coombs for instance)
Kindly post useful videos that you have...
I have completed my first read with kaplan LN for all the subjects except for pathology..i am planning to start FA with UW, my exam date on December..should i read kaplan LN for Biochemistry and for immunology, or shall i stick with FA with UW for all the subjects
because when i...
Hi guys, please i would like some advice about how do i have to study Complement in Immuno, Kaplan emphasizes on the functions and the differences between them but ive heard people talking about studying complement with najeeb lectures which gives complement very detailed, so the question is...
why is c3 level low in certain diseases, such as rheumatoid arthritis, type III hypersensitivity?
can any of you tell me all the possible diseases and the mechanisms of decreased C3?
really appreciate it! :sorry:
Can someone explain if conjugated vaccines (like HiB) activate T cells? I thought that the whole point of conjugation is to induce T cells, which can allow class switching and generate long term antibodies. If the vaccine is only activating B cells, we would only get IgM, which doesn't last...
1. brutons is B cell defect (proB cell >>>\>>> preB cell), then why there is absent thymic shadow ? as mentioned in first aid ???? 2012 edition
2. why PCP pneumonia infection is more common in HIM (hyper IgM has problem with B cell class switching, so bacterial infection not viral infection...
killed vaccine generate humoral immunity. does it involve class switching? if yes then why do we require booster as memory has already been generated!
que 2- tab( typhoid) vaccine is killed or live attenuated?
que 3- conjugated pnummococcal , flu , menningococcal are killed or live?
Why does eosinophilia occur in atypical pneumonia secondary to C. Trachomatis?
Hey guys, this is my first post here so I don't know the proper etiquette for asking questions - just let me know if there's any more information I should include in the future. Can anyone explain this to me (this is...
Why dont these patients develope graft vs host disease?
They dont have MHC II > no CD4 cells and also weak CD8 function.. In Graft vs host the graft attacks the patuent.. What's thr decrease in CD 4 has to do with the grafts ability to attack the recipient?