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Well, the answer for this is yes. However I don't get why terbutaline causes hyperglycemia when beta2 agonists increase insulin.

The explanation I found is that terbutaline increases insulin resistance. However, hypokalemia, another S/E of terbutaline is caused by increasing insulin, resulting in inward movement of K+ into cells. In this case glucose moves together with K+ so alll the situations seem to cause hypoglycemia.
Can anyone explain?
 

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liver b2 or b1 then cAMP then glucose release?

glucagon and b1-b2 agonists increase insulin release due to cAMP mediated increase in Ca then degranulation of islets. But insulin inhibits glucagon release.
 
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