[im99]

heyy very nice explanation.

i have a confusion.. how does UMN lesion result in lower limb weakness and LMN lesion result in upper limb weakness? eg. ALS.. kaplan says both upper and lower motor affected.. so FLACCID paralysis of upper limbs (so means LML upper limbs) and similarly SPASTIC paralysis of lower limbs ( so UML lower limbs).. the same thing applies to ASA occlusion also.

i thought UMN in lower limb supply the flexors more than extensors.. but cant really understand the application.. very confused.

 

[im99]

and regarding the pain in tabes dorsalis. in kaplan lectures they say that.. 'pain and temperature fibers are also irritated-- patient perceives touch in lower limb as painful.'