I've seen a UW question talking about reninomas which I did not read in Kaplan Lecture Notes or FA.
Can anybody enlighten me please
Can anybody enlighten me please
glucocorticoid mediated primary hyperaldosteronism? mechanism?The idea now is :
1) you have increased renin which in turn increase the aldosterone so now you have both increased renin and aldosterone which is called 2ry hyperaldosteronism
2 ) DD :
1ry hyperaldosteronism ( with low renin) :
adrenal adenoma ( conns syndrome ) , hyperplasia , Glucocort icoid mediated
2ry hyperaldosteronism( with high renin):
renin secrating tumour, Renal artery stenosis , Cirrhosis , Hf , CRF , Chronic laxatives use , Hypothyroidism
This thread may help explain your questionglucocorticoid mediated primary hyperaldosteronism? mechanism?![]()
How can I tell the difference between a reninoma from renovascular hypertension just by labs??? both have high renin, HT...Causes of secondary hyperaldosteronism include:
- Renovascular hypertension - renal artery stenosis
- Low volume states (congested liver in heart failure is an example)
- Reninomas which secrete renin autonomously from the JG apparatus