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Good post, but apparently the theory has been updated now

Eosinophils (attracted by ECF-A in mast cell granules) are now believed to OVERALL increase and maintainthe inflammatory response. That's why you would still warrant corticosteroid use.

Because Eosinophils release competing factors:
1) arylsulfatase and histaminase - decrease inflammation by breakdown of inflamm factors e.g. histamine (as quratulain helpfully mentioned, and still a good mnemonic imo).
2) LTC4 and PAF - stim mast cells to release more cytokines - this wins out.

actually the answer is in histology.
mast cells and basophils...and they granulate in response to FC portions of antibodies cross linkages.
The granules secreate excessive amounts of the inflammotory promoter HISTAMINE...
SO...there has to be something to deactivate the HISTAMNE....
HISTAMINASES are present in the granules of EIOSINOPHIlic granules.
hence in Allergy, and hypersensitvity...eiosinophils are active to counteract and limit the inflamation, in this eiosinophil s persist in such patients even if they are no in active allery or asthma...etc...
you can check this in whethers, the atlas...they have cool micrographs...
let me know if youre not clear...
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