On a uworld question it states in the explanation that "Additionally, blockade of Beta 2 adrenergic receptors inhibits hepatic gluconeogenesis and peripheral glycogenolysis and lipolysis. For this reason, non-selective Beta-blockers should not be used in diabetic patients."
From this I take it that it is saying if you were to STIMULATE beta 2 then you would increase hepatic gluconeogenesis and peripheral glycogenolysis --> increasing blood sugar.
In First AID 2014 on page 244 it says that Beta 2 increases insulin release. Wouldn't the insulin release due to beta 2 receptor stimulation lower hepatic gluconeogenesis and glycogenolysis and promote storage and metabolism instead?
The QID for this is 1492 if anyone wants to look it up. Please clear this up for me if you can. I understand the answer to the question, but that specific part of the explanation confused me a bit. Thanks!
From this I take it that it is saying if you were to STIMULATE beta 2 then you would increase hepatic gluconeogenesis and peripheral glycogenolysis --> increasing blood sugar.
In First AID 2014 on page 244 it says that Beta 2 increases insulin release. Wouldn't the insulin release due to beta 2 receptor stimulation lower hepatic gluconeogenesis and glycogenolysis and promote storage and metabolism instead?
The QID for this is 1492 if anyone wants to look it up. Please clear this up for me if you can. I understand the answer to the question, but that specific part of the explanation confused me a bit. Thanks!